The injection of human chorionic gonadotrophin (hcg) may provoke a marked hyperplasia of certain structures appearing in the endometrial glandular cells at the beginning of the secretory period in women. The determinant factor in this hyperplasia would seem to be the excessive hormonal secretion of the ovaries provoked by hcg, in particular, the relative hypersecretion of progesterone.
The structures studied show a clearly marked cyclic evolution; certain of them present particular regressive forms, which the authors describe and interpret. Finally, their metabolic role is discussed, especially with regard to the relationship between glycogen and certain enlarged mitochondria.
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